Wednesday, October 5, 2016

Lumigan 0.3mg / ml eye drops, solution





1. Name Of The Medicinal Product



LUMIGAN 0.3 mg/ml eye drops, solution


2. Qualitative And Quantitative Composition



One ml of solution contains 0.3 mg bimatoprost.



Excipient:



One ml of solution contains 0.05 mg benzalkonium chloride.



For a full list of excipients, see section 6.1.



3. Pharmaceutical Form



Eye drops, solution.



Colourless to slightly yellow solution.



4. Clinical Particulars



4.1 Therapeutic Indications



Reduction of elevated intraocular pressure in chronic open-angle glaucoma and ocular hypertension in adults (as monotherapy or as adjunctive therapy to beta-blockers).



4.2 Posology And Method Of Administration



The recommended dose is one drop in the affected eye(s) once daily, administered in the evening. The dose should not exceed once daily as more frequent administration may lessen the intraocular pressure lowering effect.



If more than one topical ophthalmic medicinal product is being used, each one should be administered at least 5 minutes apart.



Use in children and adolescents (under the age of 18):



LUMIGAN is not recommended for use in children below 18 years, due to a lack of data on safety and efficacy.



Use in hepatic and renal impairment:



LUMIGAN has not been studied in patients with renal or moderate to severe hepatic impairment and should therefore be used with caution in such patients. In patients with a history of mild liver disease or abnormal alanine aminotransferase (ALT), aspartate aminotransferase (AST) and/or bilirubin at baseline, bimatoprost 0.3 mg/ml eye drops, solution had no adverse effect on liver function over 24 months.



4.3 Contraindications



Hypersensitivity to the active substance or to any of the excipients.



4.4 Special Warnings And Precautions For Use



Before treatment is initiated, patients should be informed of the possibility of eyelash growth, darkening of the eyelid skin and increased iris pigmentation since these have been observed during treatment with LUMIGAN. Some of these changes may be permanent, and may lead to differences in appearance between the eyes when only one eye is treated. The change in iris pigmentation occurs slowly and may not be noticeable for several months or years. At 12 months, the incidence was 1.5% and did not increase following 3 years treatment (see section 4.8). Periorbital tissue pigmentation has been reported to be reversible in some patients.



LUMIGAN has not been studied in patients with compromised respiratory function and should therefore be used with caution in such patients. In clinical studies, in those patients with a history of a compromised respiratory function, no significant untoward respiratory effects have been seen.



LUMIGAN has not been studied in patients with heart block more severe than first degree or uncontrolled congestive heart failure. There have been a limited number of spontaneous reports of bradycardia or hypotension with bimatoprost 0.3 mg/ml eye drops, solution. LUMIGAN should be used with caution in patients predisposed to low heart rate or low blood pressure



LUMIGAN has not been studied in patients with inflammatory ocular conditions, neovascular, inflammatory, angle-closure glaucoma, congenital glaucoma or narrow-angle glaucoma.



Cystoid macular oedema has been uncommonly reported (



There have been rare spontaneous reports of reactivation of previous corneal infiltrates or ocular infections with bimatoprost 0.3 mg/ml eye drops, solution. LUMIGAN should be used with caution in patients with a prior history of significant ocular viral infections (e.g. herpes simplex) or uveitis/iritis.



Bimatoprost 0.3 mg/ml eye drops, solution contains the preservative benzalkonium chloride, which may be absorbed by soft contact lenses. Eye irritation and discolouration of the soft contact lenses may also occur because of the presence of benzalkonium chloride. Contact lenses should be removed prior to instillation and may be reinserted 15 minutes following administration.



Benzalkonium chloride, which is commonly used as a preservative in ophthalmic products, has been reported to cause punctate keratopathy and/or toxic ulcerative keratopathy. Since LUMIGAN contains benzalkonium chloride, monitoring is required with frequent or prolonged use in dry eye patients or where the cornea is compromised.



4.5 Interaction With Other Medicinal Products And Other Forms Of Interaction



No interaction studies have been performed.



No interactions are anticipated in humans, since systemic concentrations of bimatoprost are extremely low (less than 0.2 ng/ml) following ocular dosing with bimatoprost 0.3 mg/ml eye drops, solution. Bimatoprost is biotransformed by any of multiple enzymes and pathways, and no effects on hepatic drug metabolising enzymes were observed in preclinical studies.



In clinical studies, LUMIGAN was used concomitantly with a number of different ophthalmic beta



Concomitant use of LUMIGAN and antiglaucomatous agents other than topical beta-blockers has not been evaluated during adjunctive glaucoma therapy.



4.6 Pregnancy And Lactation



Pregnancy



There are no adequate data from the use of bimatoprost in pregnant women. Animal studies have shown reproductive toxicity at high maternotoxic doses (see section 5.3).



LUMIGAN should not be used during pregnancy unless clearly necessary.



Lactation



It is unknown whether bimatoprost is excreted in human breast milk. Animal studies have shown excretion of bimatoprost in breast milk. A risk to the suckling child cannot be excluded. A decision on whether to continue/discontinue breast-feeding or to continue/discontinue therapy with LUMIGAN should be made, taking into account the benefit of breast-feeding to the child and the benefit of LUMIGAN therapy to the woman.



4.7 Effects On Ability To Drive And Use Machines



LUMIGAN has negligible influence on the ability to drive and use machines. As with any ocular treatment, if transient blurred vision occurs at instillation, the patient should wait until the vision clears before driving or using machines.



4.8 Undesirable Effects



In clinical studies, over 1800 patients have been treated with LUMIGAN 0.3 mg/ml eye drops, solution. On combining the data from phase III monotherapy and adjunctive LUMIGAN 0.3 mg/ml eye drops, solution usage, the most frequently reported treatment-related adverse events were: growth of eyelashes in up to 45% in the first year with the incidence of new reports decreasing to 7% at 2 years and 2% at 3 years, conjunctival hyperaemia (mostly trace to mild and thought to be of a non-inflammatory nature) in up to 44% in the first year with the incidence of new reports decreasing to 13% at 2 years and 12% at 3 years and ocular pruritus in up to 14% of patients in the first year with the incidence of new reports decreasing to 3% at 2 years and 0% at 3 years. Less than 9% of patients discontinued due to any adverse event in the first year with the incidence of additional patient discontinuations being 3% at both 2 and 3 years.



The following adverse reactions were reported during clinical trials with LUMIGAN 0.3 mg/ml eye drops, solution. Most were ocular, mild to moderate, and none was serious:



Very common (














































System Organ class




Frequency




Undesirable effect




Nervous system disorders




common




headache




 



 




uncommon




dizziness




 




 



 




 



 




Eye disorders



 




very common




conjunctival hyperaemia, ocular pruritus, growth of eyelashes




 



 




common




superficial punctate keratitis, corneal erosion, ocular burning, ocular irritation, allergic conjunctivitis, blepharitis, worsening of visual acuity, asthenopia, conjunctival oedema, foreign body sensation, ocular dryness, eye pain, photophobia, tearing, eye discharge, visual disturbance, increased iris pigmentation, eyelash darkening.



 



 




 



 




uncommon




retinal haemorrhage, uveitis, cystoid macular oedema, iritis, blepharospasm, eyelid retraction




 




not known




enophthalmos




Vascular disorders




common




hypertension




Gastrointestinal disorders



 




uncommon




nausea




Skin and subcutaneous tissue disorders



 




common




eyelid erythema, eyelid pruritus, pigmentation of periocular skin




 



 




uncommon




eyelid oedema, hirsutism.




General disorders and administration site conditions



 




uncommon




asthenia




Investigations




common




liver function test abnormal



4.9 Overdose



No case of overdose has been reported, and is unlikely to occur after ocular administration.



If overdose occurs, treatment should be symptomatic and supportive. If LUMIGAN is accidentally ingested, the following information may be useful: in two-week oral rat and mouse studies, doses up to 100 mg/kg/day did not produce any toxicity. This dose expressed as mg/m2 is at least 70



5. Pharmacological Properties



5.1 Pharmacodynamic Properties



Pharmacotherapeutic group: Ophthalmologicals, prostaglandin analogues, ATC code: S01EE03.



The mechanism of action by which bimatoprost reduces intraocular pressure in humans is by increasing aqueous humour outflow through the trabecular meshwork and enhancing uveoscleral outflow. Reduction of the intraocular pressure starts approximately 4 hours after the first administration and maximum effect is reached within approximately 8 to 12 hours. The duration of effect is maintained for at least 24 hours.



Bimatoprost is a potent ocular hypotensive agent. It is a synthetic prostamide, structurally related to prostaglandin F (PGF), that does not act through any known prostaglandin receptors. Bimatoprost selectively mimics the effects of newly discovered biosynthesised substances called prostamides. The prostamide receptor, however, has not yet been structurally identified.



During 12 months' monotherapy treatment with LUMIGAN 0.3 mg/ml in adults, versus timolol, mean change from baseline in morning (08:00) intraocular pressure ranged from



In a 6-month clinical study with LUMIGAN 0.3 mg/ml, versus latanoprost, a statistically superior reduction in morning mean IOP (ranging from -7.6 to -8.2 mmHg for bimatoprost versus –6.0 to



Compared to treatment with beta-blocker alone, adjunctive therapy with beta-blocker and LUMIGAN 0.3 mg/ml lowered mean morning (08:00) intraocular pressure by



Limited experience is available in patients with open-angle glaucoma with pseudoexfoliative and pigmentary glaucoma, and chronic angle-closure glaucoma with patent iridotomy.



No clinically relevant effects on heart rate and blood pressure have been observed in clinical trials.



5.2 Pharmacokinetic Properties



Bimatoprost penetrates the human cornea and sclera well in vitro. After ocular administration in adults, the systemic exposure of bimatoprost is very low with no accumulation over time. After once daily ocular administration of one drop of LUMIGAN 0.3 mg/ml to both eyes for two weeks, blood concentrations peaked within 10 minutes after dosing and declined to below the lower limit of detection (0.025 ng/ml) within 1.5 hours after dosing. Mean Cmax and AUC 0-24hrs values were similar on days 7 and 14 at approximately 0.08 ng/ml and 0.09 ng•hr/ml respectively, indicating that a steady bimatoprost concentration was reached during the first week of ocular dosing.



Bimatoprost is moderately distributed into body tissues and the systemic volume of distribution in humans at steady-state was 0.67 l/kg. In human blood, bimatoprost resides mainly in the plasma. The plasma protein binding of bimatoprost is approximately 88%.



Bimatoprost is the major circulating species in the blood once it reaches the systemic circulation following ocular dosing. Bimatoprost then undergoes oxidation, N-deethylation and glucuronidation to form a diverse variety of metabolites.



Bimatoprost is eliminated primarily by renal excretion, up to 67% of an intravenous dose administered to healthy adult volunteers was excreted in the urine, 25% of the dose was excreted via the faeces. The elimination half-life, determined after intravenous administration, was approximately 45 minutes; the total blood clearance was 1.5 l/hr/kg.



Characteristics in elderly patients:



After twice daily dosing of LUMIGAN 0.3 mg/ml, the mean AUC0-24hr value of 0.0634 ng•hr/ml bimatoprost in the elderly (subjects 65 years or older) were significantly higher than 0.0218 ng•hr/ml in young healthy adults. However, this finding is not clinically relevant as systemic exposure for both elderly and young subjects remained very low from ocular dosing. There was no accumulation of bimatoprost in the blood over time and the safety profile was similar in elderly and young patients.



5.3 Preclinical Safety Data



Effects in non-clinical studies were observed only at exposures considered sufficiently in excess of the maximum human exposure indicating little relevance to clinical use.



Monkeys administered ocular bimatoprost concentrations of



Bimatoprost was not mutagenic or carcinogenic in a series of in vitro and in vivo studies.



Bimatoprost did not impair fertility in rats up to doses of 0.6 mg/kg/day (at least 103



6. Pharmaceutical Particulars



6.1 List Of Excipients



Benzalkonium chloride



Sodium chloride



Sodium phosphate dibasic heptahydrate



Citric acid monohydrate



Hydrochloric acid or sodium hydroxide (to adjust pH)



Purified water



6.2 Incompatibilities



Not applicable.



6.3 Shelf Life



2 years.



4 weeks after first opening.



6.4 Special Precautions For Storage



This medicinal product does not require any special storage conditions.



6.5 Nature And Contents Of Container



White opaque low density polyethylene bottles with polystyrene screw cap. Each bottle has a fill volume of 3 ml.



The following pack sizes are available: cartons containing 1 or 3 bottles of 3 ml solution. Not all pack sizes may be marketed.



6.6 Special Precautions For Disposal And Other Handling



No special requirements.



7. Marketing Authorisation Holder



Allergan Pharmaceuticals Ireland



Castlebar Road



Westport



Co. Mayo



Ireland



8. Marketing Authorisation Number(S)



EU/1/02/205/001-002



9. Date Of First Authorisation/Renewal Of The Authorisation



8 March 2002 / 20 February 2007



10. Date Of Revision Of The Text



7 January 2010



Detailed information on this product is available on the website of the European Medicines Agency (EMEA): http://www.emea.europa.eu





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